...小鼠，野生型小鼠，DSS结肠炎，细胞因子表达，T细胞增殖，qRT-PCR [gap=2459]Key words: IL2 heterozygous mice, wild type mice, DSS colitis, cytokine expression, qRT-PCR, T cell proliferation ..
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wild-type mice 野生型小鼠
However,as discrepant evidence, no difference in ischemic infarcts has been found between 5-LOX-deficient and wild type mice with focal cerebral ischemia.
然而，另有研究发现，5-LOX基因敲除小鼠脑缺血后脑梗塞体积同野生型小鼠相比，没有明显差别。
参考来源 - 5·2,447,543篇论文数据，部分数据来源于NoteExpress
Results Compared with wild type mice, there was no significant difference in CBF, CPS and ATP levels in L1 KO mice(P>0.05).
结果与野生型小鼠相比，L1 KO小鼠局部脑血流量、ATP含量和蛋白质合成率差异均无统计学意义（均P>0.05）。
Moreover, pre-treatment of wild type mice with a nitric oxide (. NO) synthase inhibitor prevented the HBO2-induced elevation in stem cell factor and circulating stem cells.
而且，野生型小鼠用NO合酶抑制剂预处理后阻止了HBO2介导的干细胞因子和外周血干细胞的增加。
The authors found that mice lacking CD24, a membrane protein expressed by immune and stem cells, developed an inflammatory response that was more powerful and lethal than that in wild-type mice.
作者们发现，缺乏CD 24，这种在免疫细胞和干细胞上表达的膜蛋白的老鼠，产生了比野生型老鼠更强且更致命的炎症反应。
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