脊髓损伤后胶质瘢痕形成中补体的作用_文章爱好者原创文学 关键词】 脊髓损伤 补体 胶质瘢痕 [gap=616]Key words:spinal cord injury;complement;glial scar
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研究表明,在PTPo 缺失的小鼠视网膜中心细胞中,能够通过神经胶质疤(glial scar)向外延伸的视 网膜中心细胞数量明显增多。坐骨神经损伤模型试验证明,在PTPo缺失的情况 下,神经再生速率显著提高。
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Glial scar and Inhibit molecules 痕及抑制性分子
The formation of necrosis cavum and glial scar after spinal cord injured was one of important factor that affected regeneration of nerve fiber and neuron.
脊髓损伤后所形成的坏死空洞和反应性增生的胶质瘢痕是导致脊髓损伤再生修复受阻的重要因素。
参考来源 - 人发角蛋白促进成年大鼠脊髓损伤修复的实验研究For a long time,it was considered that the central neurons would die nonreversibly after ischemic injury and be replaced by gliocytes which formed the glial scar,and then lost their function.
长期以来,人们认为中枢神经损伤后引起神经细胞不可逆的死亡,损伤的神经元只能被胶质细胞所填充,形成胶质疤痕,从而丧失了神经功能。
参考来源 - 慢病毒载体介导血管生成素·2,447,543篇论文数据,部分数据来源于NoteExpress
以上来源于: WordNet
The formation of necrosis cavum and glial scar after spinal cord injured was one of important factor that affected regeneration of nerve fiber and neuron.
脊髓损伤后所形成的坏死空洞和反应性增生的胶质瘢痕是导致脊髓损伤再生修复受阻的重要因素。
After brain damage, the expression of neurocan obviously increases again in the glial scar. It is considered that neurocan can prevent the regeneration of axon.
脑损伤后,神经蛋白聚糖在胶质瘢痕中的表达明显增加,据认为可阻止轴突再生。
After sacrifice, spinal cord sections were analyzed for MSC graft size, tissue sparing, host immune response, and glial scar formation, using specific antibodies and Nissl-myelin staining.
处死后,用特殊的抗体和髓鞘尼氏染色法处理脊髓切片,分析骨髓基质细胞移植物大小、组织保留情况、宿主免疫反应和神经胶质疤痕形成情况。
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