...alomyelitis; excitotoxicity; microglia; TNFα [gap=4091]关键词:AMPA受体;Arc/Arg3.1;实验性自身免疫性脑脊髓炎;兴奋性毒性;小胶质细胞;肿瘤坏死因子 ...
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... evidence based medicine, EBM循征医学 excitatory toxicity兴奋性毒性 exhaustion stage衰竭期 ...
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The mechanism might be related with abating the cerebral edema, dilating the cerebrovascular, inhibiting excitatory toxicity, abating the apoptosis in the ischemic semi-dark band, regulating the expression of the promoting /resisting apoptosis gene.
其机理可能与其减轻脑水肿、扩张脑血管、改善血液循环状态、缓解兴奋性毒性、抑制缺血半暗带细胞凋亡，调节促/抗凋亡基因表达有关。
参考来源 - 治疗中风病（缺血性）方剂的筛选及其作用机理的实验研究N-methyl-D-aspartate (NMDA) receptors, which belong to the ionotropic glutamate receptors, play major roles in synaptic transmission and plasticity, as well as excitotoxicity.
NMDA受体属于离子型谷氨酸受体，它在突触传递，突触可塑性及兴奋性毒性中都扮演着重要的角色。
参考来源 - NMDA受体亚单位装配、运输以及膜表面表达的分子机制·2,447,543篇论文数据，部分数据来源于NoteExpress
雌激素能拮抗兴奋性氨基酸毒性，稳定细胞内钙离子溶度。
Estrogen can resist the excitability amino acid toxicity and stable the solubility of calcium ion in cells.
结论：癫痫急性发作期n 1、P 1波的改变与兴奋性氨基酸的神经元毒性作用及继发性缺血、缺氧致神经元损伤有关。
Conclusion: the changes of N1P1 in acute attack period are related with the neuron toxic action of excitatory amino acids and lesions made by secondary ischemia and hypoxia.
因谷氨酸代谢异常，过度释放和对突触后受体的高频刺激，引起了兴奋性神经毒性，导致神经元死亡。
The abnormal metabolism and excess release of Glutamic acid as well as high-frequency stimulation to the postsynaptic receptor induce excitatory neural toxicity, resulting in the death of neurons.
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