这往往被视为是最后阶段的阿兹海默氏症。
This is often seen as the final stage of Alzheimer's disease.
一项去年发表在《阿兹海默氏症期刊》的研究发现，每天喝一点咖啡能够降低25%帕金森发病率。 。
Drinking ga few cups of coffee a day could lower the risk of developing Parkinson's disease by as much as 25 percent, according to a study published last year in the Journal of Alzheimer's Disease.
上述研究从2007年开始，一直持续到2012年，是针对轻度至中度阿兹海默氏症患者规模最大、时间最长的研究之一。
The study, which ran from 2007 to 2012, is one of the largest and longest surveys of people with mild to moderate Alzheimer's disease.
另外，去年发表于《阿兹海默氏症与痴呆症》（Alzheimer’s and Dementia）杂志的一项研究发现，使用Omega-3DHA补充品可以让大脑年轻三岁，提高记忆力和学习能力。
Plus, a study published last year in the journal Alzheimer’s and Dementia found that taking Omega-3 DHA supplements took three years off the brain, improving memory and learning skills.
阿尔兹·海默氏症是痴呆症最常见的一种，影响着整个英国约700 000人口，这个数字预计在2025年会加倍。
Alzheimer's is the most common form of dementia, which affects around 700, 000 people in the UK. That figure is expected to double by 2025.
玛格丽特·盖兹，是洛杉矶南加州大学的心理学家，她正在研究患上阿尔茨海默氏症的原因。
Margaret GatzMargaret Gatz, a psychologist at the University of Southern California, Los Angeles, is investigating the causes of Alzheimer's disease.
因为在这个实验中的小鼠已经被引发了阿尔兹·海默氏症的特征，它们被有计划的降低GCSF。
Since the mice in the experiment had been triggered to develop Alzheimer's symptoms, they were projected to show a decrease in the GCSF.
虽然还没有已经治愈阿尔兹·海默氏症的先例存在，但是实验已经在证明这种复合物与咖啡因共同作用增加了抵御阿尔兹·海默氏症发生的风险概率，这会对治疗方法的探索有些用处。
No cure for Alzheimer's exists, but identifying the compound in coffee that interacts with caffeine to create this added protection against Alzheimer's would be useful in developing treatments.
阿尔兹·海默氏症主要是由于脑部聚集结块的异常蛋白质，名为be ta淀粉状蛋白斑。
A key aspect of Alzheimer's is sticky clumps of an abnormal protein in the brain called beta amyloid plaques.
谨慎地活化这些促使神经新生的分子，也许能弥补因阿兹·海默症与帕金森氏症而死亡的神经细胞。
Careful reactivation of the molecules that foster such neurogenesis might counter the death of neurons that occurs in Alzheimer's and Parkinson's disease.
谨慎地活化这些促使神经新生的分子，也许能弥补因阿兹·海默症与帕金森氏症而死亡的神经细胞。
Careful reactivation of the molecules that foster such neurogenesis might counter the death of neurons that occurs in Alzheimer's and Parkinson's disease.
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