结论长托宁通过抑制肺血管内皮细胞ICAM - 1的表达，对大鼠脓毒症肺损伤具有保护作用。
Conclusion PQN inhibits the expression of ICAM-1 in lung vascular endothelium, and had protective effects on acute lung injury induced by sepsis.
作者研究了精制大肠杆菌内毒素对体外培养的小牛肺血管内皮细胞的影响及654-2的保护作用。
The effect of E. coli endotoxin and the protective action of anisodamine (654-2) on the bovine pulmonary endothelial cell (BPEC) in vitro were investigated.
目的探讨高浓度氧对新生鼠肺组织血管内皮生长因子(VEGF)蛋白表达及肺血管内皮细胞超微结构影响的动态变化规律。
Objective: To explore the dynamic changes of the expression of VEGF protein in the lung and ultrastructure of pulmonary vascular endothelial cell in neonatal rats with hyperoxia.
目的探讨体外循环(CPB)期间低温改良低钾右旋糖酐(LPD)溶液肺动脉灌注对CPB中肺血管内皮细胞损伤的保护作用。
Objective To test the effect of pulmonary artery perfusion with modified low-potassium dextran (LPD) solution on lung vascular endothelial cell injury during cardiopulmonary bypass (CPB).
结论雌激素水平明显影响雌鼠心脏组织和肺血管内皮细胞中雌激素受体的含量，高脂饮食可明显降低组织和细胞中雌激素受体的含量。
Conclusions the content of er in VEC and hearts are affected by serum estrogen level, and cholesterol rich diet decrease the content of er remarkably.
肺微血管内皮细胞(PMVEC)体外培养法的建立对于研究吸入性损伤具有重要意义。
The establishment of the method to culture pulmonary microvascular endothelial cells (PMVECs) is important for the study of inhalation injury.
本实验研究了654-2对油酸所致离体血管内皮细胞直接损伤作用的影响，以探讨654-2改善油酸所致急性肺损伤的机制。
The effect of anisodamin (654-2) on oleic acid-induced injury of cultured bovine endothelial cells was studied to elucidate the mechanism of protective roles of anisodamin on acute lung injury.
观察哮喘豚鼠模型嗜酸细胞和淋巴细胞与肺微血管内皮细胞的粘附率；
To explore the adhesion of peripheral blood lymphocytes and eosinophils and lung microvascular endothelial cells (LMEC) in asthma.
目的：研究小鼠肺微血管内皮细胞分离培养方法并探讨脂多糖(LPS)诱导小鼠肺微血管内皮细胞血管内皮生长因子(VEGF)表达。
Objective: To establish a isolation and cultivation method of mice lung microvessel endothelial cell (LMVEC) and investigate the VEGF expression of mice LMVEC induced by lipopolysaccharide (LPS).
结果显示，肺毛细血管内皮细胞及其细胞紧密连接明显破坏，其对硝酸镧及甲基丙烯酸甲酯的通透性显著增加。
The results showed that the capillary endothelium and their tight junctions were partly destroyed and their permeability were significantly increased to lanthanum nitrate and methyl methacrylate.
结论间断肺通气可通过减少白细胞与血管内皮的黏附，减轻肺部炎症反应、内皮细胞损伤等，对CPB所致的肺损伤有保护作用。
Conclusion Intermittent ventilation has the protective effects on lung injury during CPB by decreasing granulocytes adhesion and alleviating lung inflammatory reaction and endothelial cells injury.
结论间断肺通气可通过减少白细胞与血管内皮的黏附，减轻肺部炎症反应、内皮细胞损伤等，对CPB所致的肺损伤有保护作用。
Conclusion Intermittent ventilation has the protective effects on lung injury during CPB by decreasing granulocytes adhesion and alleviating lung inflammatory reaction and endothelial cells injury.
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