他们将原癌基因诱导的淋巴瘤移植到野生型小鼠，然后在淋巴瘤细胞启动p53表达。
They transplanted Myc-driven lymphoma cells into wild-type mice and then turned on p53 expression in the lymphoma cells.
而且，野生型小鼠用NO合酶抑制剂预处理后阻止了HBO2介导的干细胞因子和外周血干细胞的增加。
Moreover, pre-treatment of wild type mice with a nitric oxide (. NO) synthase inhibitor prevented the HBO2-induced elevation in stem cell factor and circulating stem cells.
经蛋白免疫印记法证实，与同窝野生型小鼠相比，转基因小鼠的脑组织和肌肉组织中TRPV1的表达显著升高。
As detected by Western blotting, TRPV1 expressions in the brain and skeletal muscle of the transgenic mice were significantly higher than the wild-type littermates.
结果与野生型小鼠相比，L1KO小鼠局部脑血流量、ATP含量和蛋白质合成率差异均无统计学意义（均P>0.05）。
Results Compared with wild type mice, there was no significant difference in CBF, CPS and ATP levels in L1 KO mice(P>0.05).
实验结果发现，野生型小鼠感染后，血清和脑中皆可以测得介白素- 6的增加，且增加的趋势与小鼠疾病严重程度及死亡率成正比。
Our results show that IL-6 was induced in the sera and brains and positively correlated with the mortality rate and disease severity of infected mice.
与野生型相比，这些小鼠显示出饮食抵触，体重增加和肥胖明显减少。
These mice displayed resistance to the diet, gaining significantly less weight and body fat than wild-type mice.
本文报道了在克隆了小鼠酪氨酸酶基因的基础上，在大肠杆菌表达体系中诱导表达野生型和变异型酪氨酸酶的结果。
Here we reported the cloning of the gene from murine tyrosinase, the expression of wild tyrosinase and variants in E. coli.
方法：给正常野生型C57BL/6小鼠皮下接种小鼠淋巴瘤EL4细胞，建立荷EL4肿瘤的小鼠模型。
METHODS: Mouse lymphoma EL4 cells were inoculated subcutaneously into wild type C57BL/6 mice (immune-competent mice).
方法：给正常野生型C57BL/6小鼠皮下接种小鼠淋巴瘤EL4细胞，建立荷EL4肿瘤的小鼠模型。
METHODS: Mouse lymphoma EL4 cells were inoculated subcutaneously into wild type C57BL/6 mice (immune-competent mice).
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