The combination of HGFs may accelerate the formation of the colonies of CFU GM.
同时，结果还显示，造血生长因子的联合应用可加速CFU-GM集落的形成。
The mean CFU GM colony yield was decreased and cluster yield increased in the MDS patients.
MDS患者骨髓细胞体外cfu GM集落产率减少，集簇产率增加。
Methods: Phenotype of T lymphocytes in bone marrow and CFU GM yields of bone marrow mononuclear cells with or without T cells were assayed with APAAP and semi solid culture method, respectively.
方法：用碱性磷酸酶抗碱性磷酸酶法检测再障患者骨髓中T淋巴细胞表型的变化，并用半固体培养法检测再障患者骨髓及去T细胞后骨髓单个核细胞CFU-GM集落形成情况。
The hematopoietic function was evaluated with the spleen node count under microscope, semi-solid agar CFU-GM assay and peripheral blood WBC count.
通过解剖镜下计数脾结节、半固体琼脂法检测CFU -GM及常规计数外周WBC变化进行造血功能测定。
Fetal muscle condition medium (FMCM) offers colony stimulating activity in the in vitro CFU-GM cultivation.
胎肌条件培养液(FMCM)可在CFU - GM体外培养中提供集落刺激活性。
The non-adherent cells of the cultures could generate CFU-GM for more than 6 weeks With a rapid decline in it's output as the cultures going on.
非贴壁层细胞形成CFU-GM的能力达6周以上，但随着培养时间的延长，CFU-GM的形成活性明显下降。
Serum GM index and fungal CFU in tissues were also markedly reduced in prophylactic itraconzole group.
预防性静脉滴注伊曲康唑组其血清GM含量和组织真菌负荷量比对照组明显降低。
Result showed that this therapy could inhibit CFU-L obviously, but could not inhibit CFU-GM of bone marrow of normal controls.
结果：健脾养阴泄毒法能明显抑制白血病患者CFU -L ，而对正常人骨髓CFU -GM无明显抑制作用。
Conclusion: Human hematopoietic growth factors can increase the expansion of cord blood CD34 cells and CFU-GM significantly.
结论：外源性造血生长因子可使脐血cd 34 +细胞及CFU - GM产生明显的扩增效应。
When the mice were sacrificed, tumor weight was measured, and the assay of colony forming unit-granulocyte and macrophage (CFU-GM) was performed on semi-solid culture.
裸鼠处死时测量肿瘤重量，用半固体培养基进行粒—巨噬细胞集落形成单位(CFU - GM)实验并检测血白细胞、丙氨酸转氨酶和肌酐等。
When the mice were sacrificed, tumor weight was measured, and the assay of colony forming unit-granulocyte and macrophage (CFU-GM) was performed on semi-solid culture.
裸鼠处死时测量肿瘤重量，用半固体培养基进行粒—巨噬细胞集落形成单位(CFU - GM)实验并检测血白细胞、丙氨酸转氨酶和肌酐等。
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