Signs include back pain, hind limb weakness, and paralysis.
症状有背痛、后肢无力、瘫痪等。
There was no progression of the hind limb motor function with complete spinal cord injury.
脊髓完全损伤后受试动物后肢功能无明显变化。
Conclusions Fetal spinal transplants can inhibit the apoptosis and improve rats hind limb function after SCI.
结论胚胎脊髓移植能抑制脊髓损伤后的细胞凋亡。
The pain threshold of right hind limb, sciatic nerve function index (SFI) and pathological change were observed.
观察每组右后肢的痛阈、坐骨神经功能指数(SFI)及病理改变。
In use 14 days, 28 days, using laser Doppler blood flow measurement instrument detection of ischemic hind limb perfusion.
分别于用药14天、28天时，用激光多普勒血流测定仪检测缺血后肢血流灌注量。
Objective To observe the changes of heme oxygenase 1 (HO 1) expression in the skeletal muscle after ischemia reperfusion of hind limb in rats.
目的观察大鼠缺血再灌注时骨骼肌中血红素氧合酶-1 (HO - 1)表达的变化。
The thickness and the ratio of intima area of the iliofemoral artery of the injured hind limb were decreased, while the stenosis extent was improved.
减少术侧后肢髂股动脉内膜厚度和内膜面积比率，减轻术侧后肢髂股动脉管腔狭窄程度。
Objective To observe the recovery process of hind limb function following complete spinal cord transection injury and discuss corresponding mechanism.
目的研究大鼠脊髓完全性损伤后后肢运动功能恢复的规律并探讨其机制。
Transplantation of bone marrow stem cells can improve the hind limb ischemia in the rat serum VEGF levels, and promote ischemic muscle tissue of angiogenesis.
骨髓干细胞移植可通过提高后肢缺血大鼠血清中的VEGF含量，促进缺血肌肉组织的血管新生。
Objective to investigate the feasibility of microbubble-mediated VEGF gene delivery to treat rabbit's hind limb ischemia resulted from acute artery occlusion.
目的探讨用超声微泡造影剂促进VEGF基因治疗兔下肢缺血的可行性。
In ischemic hind-limb tissue, skeletal muscle blood flow and arteriolar density increased progressively between days 2 and 21 after arterial ligation.
对于缺血的后肢组织，在动脉结扎术后的第2至第21天，骨骼肌的血流和微小动脉的密度呈进行性增加。
In ischemic hind-limb tissue, skeletal muscle blood flow and arteriolar density increased progressively between days 2 and 21 after arterial ligation.
对于缺血的后肢组织，在动脉结扎术后的第2至第21天，骨骼肌的血流和微小动脉的密度呈进行性增加。
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