Alleviate skin hyperemia and swelling.
减轻皮肤充血、浮肿、肿胀。
Various organs even hyperemia, dropsy or necrosis.
各器官均充血、水肿或坏死。
There were hyperemia, hemorrhage in lungs and pulmonary consolidation;
死亡动物肺充血、出血甚至肺部呈红肝样变；
The ulcer is shallow and sharply demarcated, with surrounding hyperemia.
溃疡表浅、边界清晰，周围有充血。
The changes mainly were edema, hyperemia and inflammatory cell infiltration.
主要病理改变为水肿、炎症细胞浸润和充血。
There are marginal hyperemia and pain, itching, burning sensation as symptoms.
睑缘充血、主觉痛痒及烧灼感。
Hyperemia and edema disappear 4 days later, chronic inflammatory cell may exist.
第4天充血水肿消退，可有慢性炎性细胞浸润。
Tan-yellow plaques are seen in the lower esophagus, along with mucosal hyperemia.
食管下段可见黄褐色的斑块，同时黏膜充血。
The infarcts seen here are yellow, with geographic borders and surrounding hyperemia.
图中的梗死灶为黄色，境界清楚，周围是充血带。
To master concept, causes, pathologic changes, and effects to body of hyperemia and congestion.
掌握充血及淤血的概念，原因，病理变化及对机体的影响。
Results:After treatment, the superficial dermis became edema and hyperemia which vanished 3 days later.
结果：治疗后见真皮浅层水肿，轻度充血。3天左右充血水肿消退或轻微。
The most common complications were conjunctival hyperemia and anterior chamber reaction, which diminished within 2 weeks.
并发症主要是前房炎症反应，在2周左右消失。
Peripheral contrast enhancement reflects hyperemia of the inflamed lymph node capsule or increased lymph node vascularity.
外周的强化反应了炎性的淋巴结包膜的充血或淋巴结血供的增多。
Hyperemia bulbar (acute red eye) : Engorgement of conjunctival blood vessels in association with acute anterior eye reaction.
球结膜充血(急性红眼)：眼前节急性反应诱发的球结膜血管充盈。
The changes of histopathology wre mainly hyperemia, congestion and edema in heart, liver, lung and kidney and injury of parenchymal cells.
病理组织学变化以心、肝、肺、肾发生充血、淤血、水肿及实质细胞发生损伤性变化为主。
Extending from the anterior portion and into the septum is a large recent myocardial infarction. The center is tan with surrounding hyperemia.
从前部直到室间隔是一大片新近梗死区，围绕中部的充血区呈棕褐色。
Signs: ocular ciliary hyperemia and punctate corneal erosion in 11 eyes, diffuse corneal opacity in 6 eyes, corneal stromal infiltration in 1 eye.
体征：眼部睫状充血，点状角膜糜烂11眼，弥漫性角膜上皮混浊6眼，角膜基质浸润1眼。
The mucosal surface of the bowel seen here shows early necrosis with hyperemia extending all the way from mucosa to submucosal and muscular wall vessels.
肠黏膜表面显示有早期坏死，从黏膜向黏膜下层和血管壁肌层都有充血。
Objective To evaluate the role of endothelium-derived hyperpolarizing factor (EDHF) in flow-dependent coronary vasodilation induced by reactive hyperemia (RH).
目的评价内皮衍生超极化因子（EDHF）在血流介导性冠状动脉反应性充血扩张中的作用。
Results The main pathological features of IC are hyperemia, edema, hemorrhage, degeneration and necrosis of bowel wall with various degrees of inflammatory reaction.
结果缺血性肠炎的病理改变以肠壁充血、水肿、出血及变性坏死为主，伴不同程度的炎症反应。
Conclusions When the brain is severely injured, there is cerebral hypoxia or ischemia during the first 24 hours, and cerebral hyperemia in the following second-fourth days.
结论重型脑损伤后24小时内存在脑缺氧、缺血，伤后第2 - 4天为脑氧合过度、脑充血。
The changing rate of the inner diameter of brachial artery after reactive hyperemia and that after held nitroglycerin in mouth were detected with high resolution ultrasound.
采用高分辨率超声测反应性充血肱动脉内径变化率和含硝酸甘油后肱动脉内径变化率。
After therapy, 32 patients (total 35 cases) feel pains is reduced, the area of hyperemia and erosion is narrowing, white stripe is decreased. The effective percentage is 91%.
结果35例糜烂型口腔扁平苔藓患者经治疗32例疼痛减轻，充血、糜烂面积缩小，白色条纹减少，有效91%。
The diameter of the brachial artery was measured by color Doppler ultrasound at rest, after reaction hyperemia and after sublingual nitroglycerin administration respectively.
应用彩色多普勒对四组之间的肱动脉进行检测，观察静息状态下、反应性充血后及含服硝酸甘油后的内径变化。
In the hyperxia solution treated group, the hyperemia, bleeding and cell infiltration in tissue interspace and alveolar cavity were obviously decreased, most alveolar walls were complete.
高氧液治疗组肺泡腔及组织间隙内充血、渗出及细胞浸润明显减少，肺泡壁多数完整。
Dietary myo-inositol deficiency in aquatic animals leads to symptoms such as a sharp decrease on the rate of growth, surface hyperemia, fin erosion, black body color, fatty liver disease.
饲料中肌醇缺乏导致水生动物生长不良、体表充血、鳍条糜烂、体色发黑、脂肪肝等症状。
Results In 21 cases, 8cases were crises of artery blood supply, the main manifestations were that capillary hyperemia reaction delay or vanish, besides the skin flap become pallor and cold;
结果21例中，动脉危象8例，主要表现为皮瓣颜色苍白、皮瓣发冷、毛细血管充盈反应迟钝或消失；
Results In 21 cases, 8cases were crises of artery blood supply, the main manifestations were that capillary hyperemia reaction delay or vanish, besides the skin flap become pallor and cold;
结果21例中，动脉危象8例，主要表现为皮瓣颜色苍白、皮瓣发冷、毛细血管充盈反应迟钝或消失；
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