Objective To study the effects and mechanism of Huangqi against myocardium injury induced by ischemia and reperfusion in rats.
目的探讨黄芪对大鼠缺血再灌注心肌的保护作用及其机制。
CONCLUSON the myocardium injury correlates with the neutrophils infiltrated in the myocardium on myocardium ischemic reperfusion in rat.
结论在大鼠心肌缺血再灌注损伤中心肌组织的损伤程度与心肌组织中浸润的中性粒细胞有密切相关性。
Objective: to explore research the protective effect of ischemia preconditioning to rabbits 'myocardium injury of ischemia reperfusion, and discuss its mechanism.
目的：从细胞保护角度出发，研究缺血预适应对家兔心肌缺血再灌注损伤的保护作用，并探讨其机制。
Objective To study the characteristics of myocardium injury which is induced by coxsackievirus B2 (CVB2) infection in the condition of selenium deficiency and adequate protein.
目的探讨柯萨奇B2病毒(CVB2)在低硒足蛋白条件下对乳鼠心肌损伤作用。
Objective To observe the protective effect of ischemic preadaptation on myocardium injury of ischemia reperfusion by adopting the animal model of ischemia reperfusion on myocardium injury rabbits.
目的采用家兔心肌缺血再灌注损伤动物模型，观察缺血预适应对心肌缺血再灌注损伤的心肌保护作用。
AIM: To study whether magnesium chloride plays a protective role in myocardium of rats with ischemia reperfusion injury, and investigate the cardioprotective action mechanism of magnesium chloride.
目的：研究氯化镁对大鼠缺血再灌注损伤心肌是否具有保护性作用，同时探讨氯化镁的心肌保护作用机制。
The activity of aspartic acid transaminase(AST) and lactic acid dehydrogenase (LDH), the area of myocardial infarction were detected for evaluation of injury degree of myocardium.
以血清天门冬氨酸转氨酶（AST）活性、乳酸脱氢酶（LDH ）活性和心肌梗死面积，评价心肌损伤程度；
Objective To elucidate the protective effect of leukocyte depletion on the myocardium during the settings of myocardial reperfusion injury.
目的研究滤除白细胞含血心肌保护液对心肌再灌注损伤的保护作用。
Results: XFK could lower the activity of enzyme of myocardium in cell-culturing fluid and alleviate ischemic injury of myocardial structure obviously.
结果：心复康可明显降低培养液中心肌酶的活性，改善心肌结构的缺血样损伤。
Myocardial injury was estimated with a semi-quantitative histological scoring system, and 11 intermediate metabolites in glycolysis and ATP, ADP, AMP, CP in myocardium were measured.
以半定量方法分析心肌病理损害程度，并测定心肌中糖酵解中间产物11项以及能量代谢产物。
Conclusion: Valerian extract can protect myocardium from ischemia reperfusion injury.
结论：缬草提取物有抗心肌缺血再灌注损伤的作用。
The results prove that ferulic acid sodium plays an important role in resisting the reperfusion injury of the ischemic myocardium.
结果初步提示：阿魏酸钠具有抗缺血心肌再灌注损伤的作用。
Conclusions: These results suggest that heparin preserve the function of myocardium after brief IR injury, the mechanism in part by promoting the production of NO, preserving the endothelium function.
结论肝素参与短暂缺血-再灌注顿抑心肌功能的保护，机制可能是通过药物性预适应，增强内皮源性NO的产生，从而减轻心肌缺血-再灌注损伤。
Conclusion: Three decoctions have various degrees of effect to injury of ischemia of myocardium, especially effect of House of Blood and Infradi-Aphragmatic Stasis-Expelling decoctions are remarkable.
结论：三逐瘀汤对心肌缺血损伤均有不同程度的保护作用，其中尤以血府逐瘀汤和膈下逐瘀汤作用明显。
The mechanism of myocardium ischemia reperfusion injury involve in generating of reactive oxygen species, overload of Ca2 + and releasing of catecholamine by ischemia.
心肌缺血再灌注损伤的主要机制为氧自由基大量释放，细胞内钙超载以及缺血导致的大量儿茶酚胺释放。
Conclusion Beating heart on cardiopulmonary bypass with mild hypothermia could attenuate ischemia-reperfusion injury of myocardium, so it has better myocardial protection effects.
结论浅低温心肺转流心脏不停跳心内直视手术可减轻心肌缺血-再灌注损伤，有较好的心肌保护作用。
This study may be indicative of the mechanism about ischemia-reperfusion injury to diabetic myocardium and the therapy for diabetic patients with ischemic heart disease.
本研究结果可能有助于提示糖尿病条件下的缺血-再灌注心肌损伤机制以及对合并缺血性心脏病的糖尿病患者的治疗方案。
Objective To investigate the protective effects of Puerarin and glucose injection (PGI) on the myocardium during myocardial ischemia and reperfusion injury (MIRI) and its mechanism.
目的探讨葛根素葡萄糖注射液(PGI)对缺血-再灌注损伤兔心肌的保护作用及其机制。
We concluded that it might be one of the important factors for the injury of myocardium that exhaustive exercise might cause the change of metabolism of thyroid hormone.
结论：力竭运动引起甲状腺激素代谢的改变是造成心肌损伤的原因之一。
Conclusion IL-8 is released during reperfusion of myocardial ischemia. It might induce infiltration and injury of neutrophils for myocardium. Its clinical significance should be further investigated.
结论IL - 8在心肌缺血和再灌注期间释放，并诱导了中性粒细胞致心肌的浸润与损伤，其在临床的意义有待进一步探讨。
It caused disturbance of myocardium metabolism and resulted in injury and necrosis.
人体免疫功能紊乱，引起心肌代谢障碍，导致心肌损伤、坏死。
The ultrastructure of myocardium showed attenuated injury.
心肌细胞超微结构表现为损伤减轻。
AIM: To investigate the changes in nuclear calcium content and permeability of nuclear pore complex in rat myocardium during ischemia reperfusion injury.
目的：探讨心肌缺血再灌注损伤时心肌细胞核钙含量与核孔复合体通透性的变化及其相互关系。
Objective To study the protective effects and mechanism of Egb on endothelial cell injury induced by myocardium experienced ischemia-reperfusion in rabbits.
目的研究银杏达莫对兔心肌缺血再灌注损伤内皮细胞的保护作用。
This paper studies amelioration of blood microcirculation and thermal injury made by 100w CO2 laser myocardium punching.
本文研究了100w CO_2激光心肌打孔对心肌组织造成的热损伤和对心肌血液微循环的改善情况。
Methods A rabbit model of myocardial ischemia and reperfusion injury was established, and HSP70mRNA in myocardium was detected by in situ hybridization histochemistry.
方法建立家兔心肌缺血再灌流损伤模型，通过原位杂交技术观察研究。
CONCLUSION: RLF may protect myocardium against ischemia injury. The mechanism may be related to the increase in activity of SOD and the suppression of the lipid peroxidation.
结论：RLF对急性缺血心肌有明显的保护作用，其作用机制可能与抑制心肌脂质过氧化，增强其抗氧化能力有关。
CONCLUSION: RLF may protect myocardium against ischemia injury. The mechanism may be related to the increase in activity of SOD and the suppression of the lipid peroxidation.
结论：RLF对急性缺血心肌有明显的保护作用，其作用机制可能与抑制心肌脂质过氧化，增强其抗氧化能力有关。
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