Objective To observe the protective effect of ischemic preadaptation on myocardium injury of ischemia reperfusion by adopting the animal model of ischemia reperfusion on myocardium injury rabbits.
目的采用家兔心肌缺血再灌注损伤动物模型，观察缺血预适应对心肌缺血再灌注损伤的心肌保护作用。
Objective: to explore research the protective effect of ischemia preconditioning to rabbits 'myocardium injury of ischemia reperfusion, and discuss its mechanism.
目的：从细胞保护角度出发，研究缺血预适应对家兔心肌缺血再灌注损伤的保护作用，并探讨其机制。
Conclusion IL-8 is released during reperfusion of myocardial ischemia. It might induce infiltration and injury of neutrophils for myocardium. Its clinical significance should be further investigated.
结论IL - 8在心肌缺血和再灌注期间释放，并诱导了中性粒细胞致心肌的浸润与损伤，其在临床的意义有待进一步探讨。
AIM: To study whether magnesium chloride plays a protective role in myocardium of rats with ischemia reperfusion injury, and investigate the cardioprotective action mechanism of magnesium chloride.
目的：研究氯化镁对大鼠缺血再灌注损伤心肌是否具有保护性作用，同时探讨氯化镁的心肌保护作用机制。
Conclusion Beating heart on cardiopulmonary bypass with mild hypothermia could attenuate ischemia-reperfusion injury of myocardium, so it has better myocardial protection effects.
结论浅低温心肺转流心脏不停跳心内直视手术可减轻心肌缺血-再灌注损伤，有较好的心肌保护作用。
Conclusion: Valerian extract can protect myocardium from ischemia reperfusion injury.
结论：缬草提取物有抗心肌缺血再灌注损伤的作用。
Objective To investigate the protective effects of Puerarin and glucose injection (PGI) on the myocardium during myocardial ischemia and reperfusion injury (MIRI) and its mechanism.
目的探讨葛根素葡萄糖注射液(PGI)对缺血-再灌注损伤兔心肌的保护作用及其机制。
This study may be indicative of the mechanism about ischemia-reperfusion injury to diabetic myocardium and the therapy for diabetic patients with ischemic heart disease.
本研究结果可能有助于提示糖尿病条件下的缺血-再灌注心肌损伤机制以及对合并缺血性心脏病的糖尿病患者的治疗方案。
Objective To study the protective effects and mechanism of Egb on endothelial cell injury induced by myocardium experienced ischemia-reperfusion in rabbits.
目的研究银杏达莫对兔心肌缺血再灌注损伤内皮细胞的保护作用。
The mechanism of myocardium ischemia reperfusion injury involve in generating of reactive oxygen species, overload of Ca2 + and releasing of catecholamine by ischemia.
心肌缺血再灌注损伤的主要机制为氧自由基大量释放，细胞内钙超载以及缺血导致的大量儿茶酚胺释放。
AIM: To investigate the changes in nuclear calcium content and permeability of nuclear pore complex in rat myocardium during ischemia reperfusion injury.
目的：探讨心肌缺血再灌注损伤时心肌细胞核钙含量与核孔复合体通透性的变化及其相互关系。
Objective To study the effects and mechanism of Huangqi against myocardium injury induced by ischemia and reperfusion in rats.
目的探讨黄芪对大鼠缺血再灌注心肌的保护作用及其机制。
Methods A rabbit model of myocardial ischemia and reperfusion injury was established, and HSP70mRNA in myocardium was detected by in situ hybridization histochemistry.
方法建立家兔心肌缺血再灌流损伤模型，通过原位杂交技术观察研究。
Methods A rabbit model of myocardial ischemia and reperfusion injury was established, and HSP70mRNA in myocardium was detected by in situ hybridization histochemistry.
方法建立家兔心肌缺血再灌流损伤模型，通过原位杂交技术观察研究。
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