Conclusion Under hypoxic conditions, propofol can cause apoptosis in PC12 cells by inducing oxidative stress injury.
结论低氧环境下丙泊酚可通过氧化应激损伤导致PC 12细胞凋亡增加。
Conclusions the increased expression of mitochondria thioredoxin reductase may protect the heart against oxidative stress injury.
结论线粒体型硫氧还蛋白还原酶基因和蛋白表达的增加有利于大鼠心肌抗氧化损伤。
Conclusion FMD has good scavenging effect to ROS, and can protect PC12 cells from oxidative stress injury induced by hydrogen peroxide.
结论FMD对氧自由基、羟自由基等ROS具有很好的清除作用，并对过氧化氢诱导的PC 12损伤具有良好的保护作用。
AIM: to observe the protective effect of melatonin MLT on the oxidative stress injury and pulmonary oedema of lung tissues in severely-burned rats.
目的：观察褪黑激素对烧伤大鼠肺组织氧化应激损伤和肺水肿的保护作用。
CONCLUSION: MLT may act an effective protection against the acute lung injury by obviously restraining oxidative stress injury and pulmonary oedema in lung tissues of severely-burned rats.
结论：褪黑激素可明显抑制烧伤后肺组织氧化应激损伤和肺水肿，对烧伤早期急性肺损伤具有一定的保护作用。
Oxidant injury, or oxidative stress, occurs when highly reactive molecules called free radicals attack and damage cellular proteins, lipids (fats) and DNA.
氧化损伤，或氧化应激，出现在被称为自由基的高活性分子攻击和破坏细胞蛋白质，脂类(脂肪)和脱氧核糖核酸时。
CONCLUSION: the illegibility of DNA and RNA fluorescent response in operation group is related with the breakage of DNA chain induced by oxidative stress during cerebral ischemia-reperfusion injury.
结论：手术组DNA和RNA荧光反射不清，可能与脑缺血再灌流损伤中氧化应激引起DNA链断裂有关。
CONCLUSION: Homocysteine can aggravate the injury of dopaminergic neurons in animal models of Parkinson disease, and the mechanism may be associated with the anti-oxidative stress.
结论：同型半胱氨酸加重离体帕金森病模型动物的多巴胺能神经元损伤，其机制可能与氧化应激反应有关。
The apoptosis in vascular endothelial cells induced by HCY plus copper was involved in the mechanism of oxidative stress-mediated injury.
HCY在生理浓度铜离子存在下可能通过氧化应激损伤的机制而导致血管内皮细胞凋亡。
This study aims to investigate the effects of oxidative stress in the pathogenesis of esophageal mucosa injury in children with reflux esophagitis (RE).
本研究主要探讨氧化应激在小儿反流性食管炎（RE）黏膜损伤中的作用机制。
Oxidative stress has been implicated in brain injury after ischemia, and the oxidants play an important role in cerebral damage.
氧化应激与缺血性脑损伤有关，氧化剂在脑损伤中起到重要作用。
In conclusion, Hyperoside effectively protected oxidative stress-induced cell injury and improved the mitochondrial function.
综上所述，金丝桃苷能够有效地保护氧化应激诱导的细胞损伤，并改善线粒体功能。
In conclusion, Hyperoside effectively protected oxidative stress-induced cell injury and improved the mitochondrial function.
综上所述，金丝桃苷能够有效地保护氧化应激诱导的细胞损伤，并改善线粒体功能。
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