Conclusions: Skeletal muscle protein degradation is increased and protein synthesis is inhibited in sepsis.
结论：感染后肌肉蛋白分解增加，并且此时蛋白合成明显受抑制。
At present, the study about the impact of IL-15 in the role of skeletal muscle protein degradation on sepsis state is rare, and the mechanism is unclear.
目前关于IL - 15对脓毒症状态下骨骼肌蛋白质分解代谢影响的研究罕见，机制尚不清楚。
Objective to explore the role of 19s regulator compound protein in the degradation of skeletal muscle protein in scalded rats.
目的探讨19s调节复合体蛋白在烫伤大鼠骨骼肌蛋白降解中的作用机制。
In essence, the maintenance of skeletal muscle mass is the result of the dynamic balance between muscle protein synthesis and muscle protein degradation.
在本质上，骨骼肌体积的保持是肌蛋白合成与降解动态平衡的结果。
Ubiquitin system, a new protein degradation pathway, is regarded as the important one for proteolysis of skeletal muscle.
泛素系统是近年来新发现的蛋白降解途径，目前研究认为该系统在骨骼肌蛋白高分解代谢中有重要作用。
Ubiquitin system, a new protein degradation pathway, is regarded as the important one for proteolysis of skeletal muscle.
泛素系统是近年来新发现的蛋白降解途径，目前研究认为该系统在骨骼肌蛋白高分解代谢中有重要作用。
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