Results Uric acid and history were risk factors of left ventricular remodeling.
结果病史和尿酸浓度是左心室重塑的主要危险因素。
Conclusion: Shenmai injection can prevent left ventricular remodeling after AMI in rats.
结论：参麦注射液能改善大鼠ami后心室重构。
ConclusionTMZ may improve left ventricular remodeling and decrease BNP level in CRF rats.
结论曲美他嗪降低血浆BNP水平，可改善慢性肾衰竭大鼠左心室重塑。
In addition, apoptosis participates in ventricular remodeling after myocardial infarction.
此外，细胞凋亡还参与急性心肌梗死后心室重构过程。
Thus, it is a key therapy to inhibit the vicious cycle of neurohormone and ventricular remodeling.
因此，阻断神经激素和心肌重构的恶性循环是心衰治疗的关键。
Reverse left ventricular remodeling has an additive role in correcting mitral valve closure, they conclude.
反响左心室重构在纠正二尖瓣闭合方面也具有价值。
Early thrombolytic therapy is beneficial to cardiac protection, cardiac function and ventricular remodeling.
早期溶栓治疗，对保护心肌、改善心功能及心室重构产生有益作用。
CONCLUSION the systolic blood pressure is one of the risk factors contributing to left ventricular remodeling.
结论收缩血压是影响左心室重构的主要危险因素。
CONCLUSION Losartan and Perindopril could improve the left ventricular remodeling and heart functions in AMI rats.
结论氯沙坦、培多普利可以减轻左室重构，改善心功能障碍。
Objective To explore some new indices for evaluation of left ventricular remodeling in dilated cardiomyopathy (DCM).
目的探讨扩张型心肌病(DCM)左心室几何重构的形态学评价新指标。
Ventricular remodeling begins in the early stage of acute myocardial infarction, with the reducing of heart function.
急性心肌梗死病人早期即可出现心室重构，心功能下降或呈下降趋势。
Preventing progressive left ventricular remodeling is a key therapeutic objective after acute myocardial infarction (MI).
防止左室重构进展是治疗急性心肌梗死(MI)的关键点。
In addition, chronic ventricular-vascular stiffness may induce ventricular remodeling and myocardial dysfunction over time.
此外，随着时间的推移，慢性心室-血管的僵硬可促进心室重构及心肌机能障碍。
Objective To observe the mechanism of action of panax quinquefolium diolsaponins (PQDS) on anti-ventricular remodeling in rats.
目的：观察西洋参叶二醇组皂苷（PQDS）对抗大鼠心室重构的作用机制。
Objective To investigate the protective effect of Panax Quinquefolium L. Diol saponin on experimental ventricular remodeling in rats.
目的观察西洋参叶二醇组皂苷对大鼠实验性心室重构的保护作用。
Objective: To investigate the effects of Shenmai injection on ventricular remodeling after acute myocardial infarction (AMI) in rats.
目的：探讨参麦注射液对急性心肌梗死(ami)大鼠心室重构的影响。
OBJECTIVE to observe the effects of captopril and low-dosage aspirin combination on left ventricular remodeling in heart failure rats.
目的：观察联用卡托普利和低剂量阿司匹林对心衰大鼠左室重建的影响。
Conclusions PCI prevented left ventricular remodeling and improved cardiac function in patients with left ventricular dysfunction and CTO.
结论P CI能够改善伴左心室收缩功能不全的CTO患者左心室收缩功能，改善左心室重构。
Therefore, the pathophysiologic process of left ventricular remodeling and to find better treatment have been better appreciated recently.
因此，研究心梗后左室重构的病理机制和探索合理的治疗措施是近年来冠心病研究的热点问题之一。
TIMP-1 may promote ventricular remodeling and accelerate the process of the myocardial diastolic dysfunction in hypertensive heart disease.
TIMP - 1可能促进了高血压患者心室重构和心肌舒张功能障碍的进程。
Objective: To evaluate the diagnostic value of BNP in the left ventricular remodeling and the predictive value of BNP in the survival after AMI.
目的：探讨急性心肌梗死后血浆脑钠肽（BNP）水平对心室重构的诊断价值及其对心肌梗死后心源性死亡的预测价值。
Objective To investigate the endothelial dysfunction and its relationship with left ventricular remodeling in patients with metabolic syndrome (MS).
目的探讨代谢综合征(MS)患者血管内皮功能与左心室重构情况及两者的相关性。
Objective To explore the relationship between blood pressure (BP) response to exercise and left ventricular remodeling in essential hypertension (EH).
目的探讨高血压患者运动血压与左室重构的关系。
Objective To study the effects of delayed percutaneous coronary interventions (PCI) on left ventricular remodeling (LVRM) after acute myocardial infarction (AMI).
目的探讨急性心肌梗死(ami)后择期经皮冠状动脉介入治疗(pci)对左心室重构(LVRM)的影响。
Background and ObjectiveTo study the effects of allogeneic bone marrow mononuclear cell transplantation on ventricular remodeling after acute myocardial infarction.
背景与目的研究急性心肌梗死后，骨髓单个核细胞梗死区移植对心室重构的影响。
Medicine, surgery and intervention operation cant substitute the putrescent tissue and prevent ventricular remodeling, Although they can improve the cardiac function.
尽管药物疗法、手术治疗和介入治疗可以改善患者的心脏功能，但是都不可替代坏死的心肌组织及阻止心肌重构的发生。
The affection to cardiomyocytes apoptosis when ACEI or ATR1A is used to therapy CHF, and its connection with left ventricular remodeling and heart function improvement.
ACEI和ATR1A类药物在治疗CHF时对心肌细胞凋亡的影响及与心室重构、心功能改善的关系。
Many studies have shown that inhibiting or attenuating cardiomyocyte apoptosis can prevent ventricular remodeling after myocardial infarction and improve cardiac function.
研究表明，抑制或减少心肌细胞凋亡能阻止心肌梗死后心室重构和改善心功能。
Conclusion: Both ABCI and RQI, though without positive myodynamia, showed certain inhibitory effect of left ventricular remodeling in rats with left heart failure after mi.
结论：无正性肌力作用的活血注射液与益气注射液对心梗后心衰大鼠左心室重构均有一定的抑制作用。
Conclusion: Both ABCI and RQI, though without positive myodynamia, showed certain inhibitory effect of left ventricular remodeling in rats with left heart failure after mi.
结论：无正性肌力作用的活血注射液与益气注射液对心梗后心衰大鼠左心室重构均有一定的抑制作用。
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