Results Compared with wild type mice, there was no significant difference in CBF, CPS and ATP levels in L1 KO mice(P>0.05).
结果与野生型小鼠相比，L1KO小鼠局部脑血流量、ATP含量和蛋白质合成率差异均无统计学意义（均P>0.05）。
Moreover, pre-treatment of wild type mice with a nitric oxide (. NO) synthase inhibitor prevented the HBO2-induced elevation in stem cell factor and circulating stem cells.
而且，野生型小鼠用NO合酶抑制剂预处理后阻止了HBO2介导的干细胞因子和外周血干细胞的增加。
The authors found that mice lacking CD24, a membrane protein expressed by immune and stem cells, developed an inflammatory response that was more powerful and lethal than that in wild-type mice.
作者们发现，缺乏CD 24，这种在免疫细胞和干细胞上表达的膜蛋白的老鼠，产生了比野生型老鼠更强且更致命的炎症反应。
Female wild-type mice on an ethanol-rich diet had 4 times as much alanine aminotransferase (ALT), indicating liver injury, than mice lacking the osteopontin gene.
雌性野生种小鼠的丙氨酸转氨酶是基因敲除鼠的4倍，这提示肝损伤的存在；
METHODS: Mouse lymphoma EL4 cells were inoculated subcutaneously into wild type C57BL/6 mice (immune-competent mice).
方法：给正常野生型C57BL/6小鼠皮下接种小鼠淋巴瘤EL4细胞，建立荷EL4肿瘤的小鼠模型。
Male wild-type mice had twice the ALT levels (P < . 05 in both cases).
雄性野生种小鼠的此数值为2倍（P <.05）。
These mice displayed resistance to the diet, gaining significantly less weight and body fat than wild-type mice.
与野生型相比，这些小鼠显示出饮食抵触，体重增加和肥胖明显减少。
They transplanted Myc-driven lymphoma cells into wild-type mice and then turned on p53 expression in the lymphoma cells.
他们将原癌基因诱导的淋巴瘤移植到野生型小鼠，然后在淋巴瘤细胞启动p53表达。
As detected by Western blotting, TRPV1 expressions in the brain and skeletal muscle of the transgenic mice were significantly higher than the wild-type littermates.
经蛋白免疫印记法证实，与同窝野生型小鼠相比，转基因小鼠的脑组织和肌肉组织中TRPV1的表达显著升高。
As detected by Western blotting, TRPV1 expressions in the brain and skeletal muscle of the transgenic mice were significantly higher than the wild-type littermates.
经蛋白免疫印记法证实，与同窝野生型小鼠相比，转基因小鼠的脑组织和肌肉组织中TRPV1的表达显著升高。
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