Results: The release of excess excitatory amino acids caused neuronal death in ischemic retina.
结果：视网膜缺血时兴奋性氨基酸过度释放导致神经元死亡。
Objective to observe the effect of excitatory amino acids receptor agonist on the onset of puberty in female rats.
目的研究兴奋性氨基酸受体激动剂对雌性大鼠青春期启动的影响。
Purpose:To study the effects of excitatory amino acids receptor noncompetitive antagonist-dextromethorphan(DM)on the traumatic brain edema.
目的：探讨兴奋性氨基酸受体非竞争性拮抗剂右甲吗喃对创作性脑水肿的防治作用。
Objective to clarify the effect of electro acupuncture (EA) on extracellular excitatory amino acids (EAA) changes during cerebral ischemia.
目的观察电针对脑缺血大鼠纹状体细胞外兴奋性氨基酸类递质水平的影响。
Conclusion Baicalin possesses protective effect on OGD injury in rat brain slices, which is related to its inhibitory effect on excitatory amino acids toxicity.
结论黄芩苷对大鼠脑片缺氧缺糖性损伤具有一定的保护作用，作用机制可能与其抑制了兴奋性氨基酸毒性有关。
Recent studies have demonstrated that free radicals, excitatory amino acids, and membrane permeability to calcium are all important factors in ischemic brain injury.
最近研究表明，自由基、兴奋性氨基酸和膜对钙的通透性是缺血性脑损伤的重要因素。
Conclusion: the changes of N1P1 in acute attack period are related with the neuron toxic action of excitatory amino acids and lesions made by secondary ischemia and hypoxia.
结论：癫痫急性发作期n 1、P 1波的改变与兴奋性氨基酸的神经元毒性作用及继发性缺血、缺氧致神经元损伤有关。
The pathophysiological mechanisms of ischemic cerebral injury involved in the release of excitatory amino acids, depolarization about the infarction, inflammatory reaction and neuron apoptosis.
缺血性脑损伤的病理生理机制包括兴奋性氨基酸的释放、梗死灶周围去极化、炎症反应及神经元凋亡等。
The pathophysiological mechanisms of ischemic cerebral injury involved in the release of excitatory amino acids, depolarization about the infarction, inflammatory reaction and neuron apoptosis.
缺血性脑损伤的病理生理机制包括兴奋性氨基酸的释放、梗死灶周围去极化、炎症反应及神经元凋亡等。
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