Ischemia and hypoxia induce the death of nerve cells and discontinue of the nerve fiber.
缺血、缺氧导致神经细胞死亡和神经纤维中断。
Three essential elements of glaucomatous optic nerve damages are optic nerve rim loss, retinal nerve fiber layer defects (RNFLD) and optic disc linear hemorrhages.
诊断青光眼视神经损害的三要素为盘沿丢失、视网膜神经纤维层缺损(RNFLD)及视盘线状出血。
CONCLUSION: Astrocytes may take part in the further lesion of nerve fiber in glaucoma, and inhibit the regeneration of nerve fiber axis-cylinder.
结论：星形胶质细胞可能参与了青光眼神经纤维的进一步损害和抑制了神经纤维轴索再生。
BACKGROUND: the environmental change of regeneration of peripheral nerve fib er can accelerate the regeneration of nerve fiber and the recovery of nerve function.
背景：周围神经纤维再生环境的改变，可以促进神经纤维的再生速度，加速神经功能的恢复。
Results it was found that nerve conduction velocity, regenerated myelinated nerve fiber cross section area and number of nerve fiber were superior to those of the contrast.
结果实验组神经传导速度、再生的有髓神经纤维横截面积、数目均优于对照组。
Glaucoma is a chronic optic neuropathy characterized by retinal ganglion cell death resulting in damage to the optic nerve head and the retinal nerve fiber layer.
青光眼是一种慢性视神经病变，它的特征是视网膜的视网膜神经节细胞死亡，造成视神经盘和视网膜神经纤维层的损害。
Glaucoma is a chronic optic neuropathy characterized by retinal ganglion cell death resulting in damage to the optic nerve head and the retinal nerve fiber layer.
青光眼是一种慢性视神经病变，它的特征是视网膜的视网膜神经节细胞死亡，造成视神经盘和视网膜神经纤维层的损害。
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