Properly inhibiting the binding of ox-LDL to LOX-1 will prevent damage from the source, and therefore protect vascular function and prevent disease occurrence.
适当阻断LOX- 1与ox - LDL的结合，将会从源头上阻止ox - LDL对内皮细胞的损伤，从而保护血管功能，防止疾病的发生。
Conclusion: Increased level of circulating ox-LDL is associated with worse fibrocalcific remodelling of valvular tissue in AS.
结论：循环氧化低密度脂蛋白水平的升高与主动脉瓣膜狭窄中的瓣膜组织纤维钙化重构有密切联系。
Objective To investigate correlation between oxidatively modified low density lipoprotein (OX-LDL), serum ferritin (SF) and the severity of coronary artery lesion.
目的探讨血浆氧化修饰低密度脂蛋白（OX -LDL）及血清铁蛋白（SF）与冠状动脉病变程度的关系。
Aim to explore the effect of oxidized low density lipoprotein (ox LDL) on cholesterol accumulation in mouse peritoneal macrophages and its relationship with activity of lysosomal cathepsin.
研究氧化型低密度脂蛋白对小鼠腹腔巨噬细胞胆固醇蓄积的影响，并探讨其与组织蛋白酶活性之间的关系。
ABSTRACT Objective To discuss the relationship between vascular endothelium function and OX-LDL change in senile patients with hypertension so as to find out better treatment.
目的探讨老年高血压患者血管内皮功能与氧化低密度脂蛋白变化关系，寻求较好的治疗方法。
Objective: To observe the effects of liquid mixture of Monascus and grape seed procyanidins extract (GSPE) on levels of cholesterol and ox-LDL of atherosclerosis rats.
目的：探讨红曲与葡萄籽原花青素(GSPE)混合剂对大鼠高脂血症的改善及对血清氧化性低密度脂蛋白的影响。
Objective to investigate the effects of oxidized low-density lipoprotein receptor 1 (LOX-1) on secretion of adhesive molecules mediated by ox-LDL in human umbilical endothelial cells (HUVECs).
目的探讨血凝素样氧化低密度脂蛋白受体1 (LOX - 1)在氧化低密度脂蛋白(ox - LDL)诱导血管内皮细胞粘附分子表达中的作用。
The standard plasma lipid profile, the size of LDL particles and the plasma level of circulating ox-LDL (4E6 antibody) were determined.
并测定标准血浆脂蛋白，低密度脂蛋白颗粒大小以及血浆中循环氧化低密度脂蛋白（4E6抗体）水平。
Conclusion BRF has dramatically reduced serum anti ox LDL antibody levels and this effect may be related to its anti atherogenesis.
这表明黑米皮的抗动脉粥样硬化作用可能与其降低抗氧化型低密度脂蛋白抗体水平有关。
ConclusionThe cell adhesion molecules and OX-LDL involves the occurrence and development of AMI, and are closely related with the severity and prognosis of the patients.
结论细胞黏附分子和OX - LDL参与了AMI的发生，且与病情严重程度和预后有一定关系。
CONCLUSION: OX-LDL enhances the expression and activity of MMP-2 in HUVEC, which may provide a explanation for the phenomenon that OX-LDL induces atherosclerotic lesions and plaque rapture.
结论：OX - LDL能促进内皮细胞表达MMP - 2，这提示血管细胞外基质的降解在氧化脂蛋白诱发的动脉粥样硬化斑块产生和破裂机制中起着一定作用。
CONCLUSIONS: OFR induces endothelial dysfunction through increasing ADMA concentration, while captopril relieves endothelial dysfunction induced by ox-LDL through decreasing ADMA concentration.
结论：OFR通过增加ADMA导致内皮功能紊乱，卡托普利则能通过减少ADMA减轻OFR诱导的内皮细胞代谢功能障碍。
LOX-1 was identified from endothelial cells as the main receptor specially recognizing and uptaking the ox-LDL, LOX-1 plays a key role in the endothelial dysfunction triggered by ox-LDL.
凝集素样氧化低密度脂蛋白受体(LOX - 1)是内皮细胞膜上特异性识别摄取ox - LDL的最主要受体，LOX - 1在介导ox - LDL导致内皮功能紊乱的过程中起关键作用。
LOX-1 was identified from endothelial cells as the main receptor specially recognizing and uptaking the ox-LDL, LOX-1 plays a key role in the endothelial dysfunction triggered by ox-LDL.
凝集素样氧化低密度脂蛋白受体(LOX - 1)是内皮细胞膜上特异性识别摄取ox - LDL的最主要受体，LOX - 1在介导ox - LDL导致内皮功能紊乱的过程中起关键作用。
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