目的：观察急性不完全脊髓损伤患者血清中MCP - 1的表达，探讨继发性脊髓损伤的可能机制。
OBJECTIVE: To observe MCP 1 expression in the serum of patients with acute spinal cord injury and explore the possible mechanism of secondary spinal cord injury.
结论脊髓伤后脊髓组织中一氧化氮(no)与eaa的释放相互促进，因而在继发性脊髓损伤中形成级联放大的神经毒性因子释放。
Conclusion no and EAA can promote each other, and result in cascade release of nervous noxious factors in secondary spinal cord injury.
目的：旨在探讨神经肽在脊髓继发性损伤中的作用。
Aim: To study the effect of neuropeptides on the secondary spinal cord injury.
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